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Sapolsky, R. M., Krey, L. C., McEwen, B. S. (1984) Glucocorticoid-sensitive hippocampal neurons are involved in terminating the adrenocortical stress response. Proceedings of the National Academy of Sciences, 81 (19) 6174-6177 doi:10.1073/pnas.81.19.6174

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Reference TypeJournal (article/letter/editorial)
TitleGlucocorticoid-sensitive hippocampal neurons are involved in terminating the adrenocortical stress response.
JournalProceedings of the National Academy of Sciences
AuthorsSapolsky, R. M.Author
Krey, L. C.Author
McEwen, B. S.Author
Year1984 (October 1)Volume81
Issue19
PublisherProceedings of the National Academy of Sciences
DOIdoi:10.1073/pnas.81.19.6174Search in ResearchGate
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Mindat Ref. ID1437129Long-form Identifiermindat:1:5:1437129:1
GUID0
Full ReferenceSapolsky, R. M., Krey, L. C., McEwen, B. S. (1984) Glucocorticoid-sensitive hippocampal neurons are involved in terminating the adrenocortical stress response. Proceedings of the National Academy of Sciences, 81 (19) 6174-6177 doi:10.1073/pnas.81.19.6174
Plain TextSapolsky, R. M., Krey, L. C., McEwen, B. S. (1984) Glucocorticoid-sensitive hippocampal neurons are involved in terminating the adrenocortical stress response. Proceedings of the National Academy of Sciences, 81 (19) 6174-6177 doi:10.1073/pnas.81.19.6174
In(1984, October) Proceedings of the National Academy of Sciences Vol. 81 (19) Proceedings of the National Academy of Sciences

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Magarinos, A. M., McEwen, B. S. (2000) Experimental diabetes in rats causes hippocampal dendritic and synaptic reorganization and increased glucocorticoid reactivity to stress. Proceedings of the National Academy of Sciences, 97 (20) 11056-11061 doi:10.1073/pnas.97.20.11056
Gupta, C., Katsumata, M., Goldman, A. S., Herold, R., Piddington, R. (1984) Glucocorticoid-induced phospholipase A2-inhibitory proteins mediate glucocorticoid teratogenicity in vitro. Proceedings of the National Academy of Sciences, 81 (4) 1140-1143 doi:10.1073/pnas.81.4.1140
MacQueen, G. M., Campbell, S., McEwen, B. S., Macdonald, K., Amano, S., Joffe, R. T., Nahmias, C., Young, L. T. (2003) Course of illness, hippocampal function, and hippocampal volume in major depression. Proceedings of the National Academy of Sciences, 100 (3) 1387-1392 doi:10.1073/pnas.0337481100
Wigdahl, B., Smith, C. A., Traglia, H. M., Rapp, F. (1984) Herpes simplex virus latency in isolated human neurons. Proceedings of the National Academy of Sciences, 81 (19) 6217-6221 doi:10.1073/pnas.81.19.6217
Hunter, R. G., Murakami, G., Dewell, S., Seligsohn, M., Baker, M. E. R., Datson, N. A., McEwen, B. S., Pfaff, D. W. (2012) Acute stress and hippocampal histone H3 lysine 9 trimethylation, a retrotransposon silencing response. Proceedings of the National Academy of Sciences, 109 (43) 17657-17662 doi:10.1073/pnas.1215810109
Dinkel, K., Dhabhar, F. S., Sapolsky, R. M. (2004) Neurotoxic effects of polymorphonuclear granulocytes on hippocampal primary cultures. Proceedings of the National Academy of Sciences, 101 (1) 331-336 doi:10.1073/pnas.0303510101
Mamounas, L. A., Thompson, R. F., Lynch, G., Baudry, M. (1984) Classical conditioning of the rabbit eyelid response increases glutamate receptor binding in hippocampal synaptic membranes. Proceedings of the National Academy of Sciences, 81 (8) 2548-2552 doi:10.1073/pnas.81.8.2548
Canady, R. A., Kroodsma, D. E., Nottebohm, F. (1984) Population differences in complexity of a learned skill are correlated with the brain space involved. Proceedings of the National Academy of Sciences, 81 (19) 6232-6234 doi:10.1073/pnas.81.19.6232
Stocker, M., Krause, M., Pedarzani, P. (1999) An apamin-sensitive Ca2+-activated K+ current in hippocampal pyramidal neurons. Proceedings of the National Academy of Sciences, 96 (8) 4662-4667 doi:10.1073/pnas.96.8.4662
Hampson, R. E., Deadwyler, S. A. (1996) Ensemble codes involving hippocampal neurons are at risk during delayed performance tests. Proceedings of the National Academy of Sciences, 93 (24) 13487-13493 doi:10.1073/pnas.93.24.13487
Sapolsky, R. M. (2004) Organismal stress and telomeric aging: An unexpected connection. Proceedings of the National Academy of Sciences, 101 (50) 17323-17324 doi:10.1073/pnas.0408041101
 
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