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Chaturvedi, Shruti, DeBaun, Michael R. (2022) Altered VWF:ADAMTS13 homeostasis is a target for therapeutic intervention in sickle cell disease. Proceedings of the National Academy of Sciences, 119 (40) doi:10.1073/pnas.2213079119

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Reference TypeJournal (article/letter/editorial)
TitleAltered VWF:ADAMTS13 homeostasis is a target for therapeutic intervention in sickle cell disease
JournalProceedings of the National Academy of Sciences
AuthorsChaturvedi, ShrutiAuthor
DeBaun, Michael R.Author
Year2022 (October 4)Volume119
Issue40
PublisherProceedings of the National Academy of Sciences
DOIdoi:10.1073/pnas.2213079119Search in ResearchGate
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Mindat Ref. ID15415422Long-form Identifiermindat:1:5:15415422:6
GUID0
Full ReferenceChaturvedi, Shruti, DeBaun, Michael R. (2022) Altered VWF:ADAMTS13 homeostasis is a target for therapeutic intervention in sickle cell disease. Proceedings of the National Academy of Sciences, 119 (40) doi:10.1073/pnas.2213079119
Plain TextChaturvedi, Shruti, DeBaun, Michael R. (2022) Altered VWF:ADAMTS13 homeostasis is a target for therapeutic intervention in sickle cell disease. Proceedings of the National Academy of Sciences, 119 (40) doi:10.1073/pnas.2213079119
In(2022, October) Proceedings of the National Academy of Sciences Vol. 119 (40) Proceedings of the National Academy of Sciences

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Archer, N. M. (2014) A diagnostic role for dense cells in sickle cell disease. Proceedings of the National Academy of Sciences, 111 (41) 14647-14648 doi:10.1073/pnas.1416147111
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Higgs, D. R., Wood, W. G. (2008) Genetic complexity in sickle cell disease. Proceedings of the National Academy of Sciences, 105 (33) 11595-11596 doi:10.1073/pnas.0806633105
Lancaster, J. R. (2002) Reaping of nitric oxide by sickle cell disease. Proceedings of the National Academy of Sciences, 99 (2) 552-553 doi:10.1073/pnas.032633399
Votano, J. R., Altman, J., Wilchek, M., Gorecki, M., Rich, A. (1984) Potential use of biaromatic L-phenylalanyl derivatives as therapeutic agents in the treatment of sickle cell disease. Proceedings of the National Academy of Sciences, 81 (10) 3190-3194 doi:10.1073/pnas.81.10.3190
Ray, S. S., Lansbury, P. T. (2004) A possible therapeutic target for Lou Gehrig's disease. Proceedings of the National Academy of Sciences, 101 (16) 5701-5702 doi:10.1073/pnas.0401934101
Wood, John C. (2016) The heart in sickle cell disease, a model for heart failure with preserved ejection fraction. Proceedings of the National Academy of Sciences, 113 (35) 9670-9672 doi:10.1073/pnas.1611899113
(2022) Correction to Supporting Information for Palmer et al., Altered cell and RNA isoform diversity in aging Down syndrome brains. Proceedings of the National Academy of Sciences, 119 (37) doi:10.1073/pnas.2212181119
(2018) Correction for Lee et al., Altered ER–mitochondria contact impacts mitochondria calcium homeostasis and contributes to neurodegeneration in vivo in disease models. Proceedings of the National Academy of Sciences, 115 (42) doi:10.1073/pnas.1815900115
(2022) Correction for Liu et al., β-Arrestin2 is a critical component of the GPCR–eNOS signalosome. Proceedings of the National Academy of Sciences, 119 (40) doi:10.1073/pnas.2213999119
Dickie, M., Serrouya, R. (2022) Picky eating as a means for coexistence. Proceedings of the National Academy of Sciences, 119 (40) doi:10.1073/pnas.2213906119
 
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