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Yoshioka, N., Wang, L., Kishimoto, K., Tsuji, T., Hu, G.-f. (2006) A therapeutic target for prostate cancer based on angiogenin-stimulated angiogenesis and cancer cell proliferation. Proceedings of the National Academy of Sciences, 103 (39) 14519-14524 doi:10.1073/pnas.0606708103

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Reference TypeJournal (article/letter/editorial)
TitleA therapeutic target for prostate cancer based on angiogenin-stimulated angiogenesis and cancer cell proliferation
JournalProceedings of the National Academy of Sciences
AuthorsYoshioka, N.Author
Wang, L.Author
Kishimoto, K.Author
Tsuji, T.Author
Hu, G.-f.Author
Year2006 (September 26)Volume103
Issue39
PublisherProceedings of the National Academy of Sciences
DOIdoi:10.1073/pnas.0606708103Search in ResearchGate
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Mindat Ref. ID1730897Long-form Identifiermindat:1:5:1730897:7
GUID0
Full ReferenceYoshioka, N., Wang, L., Kishimoto, K., Tsuji, T., Hu, G.-f. (2006) A therapeutic target for prostate cancer based on angiogenin-stimulated angiogenesis and cancer cell proliferation. Proceedings of the National Academy of Sciences, 103 (39) 14519-14524 doi:10.1073/pnas.0606708103
Plain TextYoshioka, N., Wang, L., Kishimoto, K., Tsuji, T., Hu, G.-f. (2006) A therapeutic target for prostate cancer based on angiogenin-stimulated angiogenesis and cancer cell proliferation. Proceedings of the National Academy of Sciences, 103 (39) 14519-14524 doi:10.1073/pnas.0606708103
In(2006, September) Proceedings of the National Academy of Sciences Vol. 103 (39) Proceedings of the National Academy of Sciences

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Hu, G.-f. (1998) Neomycin inhibits angiogenin-induced angiogenesis. Proceedings of the National Academy of Sciences, 95 (17) 9791-9795 doi:10.1073/pnas.95.17.9791
Xu, W., Ngo, L., Perez, G., Dokmanovic, M., Marks, P. A. (2006) Intrinsic apoptotic and thioredoxin pathways in human prostate cancer cell response to histone deacetylase inhibitor. Proceedings of the National Academy of Sciences, 103 (42) 15540-15545 doi:10.1073/pnas.0607518103
Drake, J. M., Huang, J. (2014) PIP5K1  inhibition as a therapeutic strategy for prostate cancer. Proceedings of the National Academy of Sciences, 111 (35) 12578-12579 doi:10.1073/pnas.1413363111
Hillerdal, V., Nilsson, B., Carlsson, B., Eriksson, F., Essand, M. (2012) T cells engineered with a T cell receptor against the prostate antigen TARP specifically kill HLA-A2+ prostate and breast cancer cells. Proceedings of the National Academy of Sciences, 109 (39) 15877-15881 doi:10.1073/pnas.1209042109
Greider, C. W. (1998) Telomerase activity, cell proliferation, and cancer. Proceedings of the National Academy of Sciences, 95 (1) 90-92 doi:10.1073/pnas.95.1.90
(2014) Correction for Tucci et al., Loss of p63 and its microRNA-205 target results in enhanced cell migration and metastasis in prostate cancer. Proceedings of the National Academy of Sciences, 111 (7) 2855 doi:10.1073/pnas.1324223111
Hu, G.-f., Riordan, J. F., Vallee, B. L. (1997) A putative angiogenin receptor in angiogenin-responsive human endothelial cells. Proceedings of the National Academy of Sciences, 94 (6) 2204-2209 doi:10.1073/pnas.94.6.2204
Xin, L., Teitell, M. A., Lawson, D. A., Kwon, A., Mellinghoff, I. K., Witte, O. N. (2006) Progression of prostate cancer by synergy of AKT with genotropic and nongenotropic actions of the androgen receptor. Proceedings of the National Academy of Sciences, 103 (20) 7789-7794 doi:10.1073/pnas.0602567103
Nelson, W. G. (2011) Profiling prostate cancer. Proceedings of the National Academy of Sciences, 108 (52) 20861-20862 doi:10.1073/pnas.1118444109
Collins, K., Jacks, T., Pavletich, N. P. (1997) The cell cycle and cancer. Proceedings of the National Academy of Sciences, 94 (7) 2776-2778 doi:10.1073/pnas.94.7.2776
Liu, Y., Bodmer, W. F. (2006) Analysis of P53 mutations and their expression in 56 colorectal cancer cell lines. Proceedings of the National Academy of Sciences, 103 (4) 976-981 doi:10.1073/pnas.0510146103
 
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