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Gray, D. H. D., Gavanescu, I., Benoist, C., Mathis, D. (2007) Danger-free autoimmune disease in Aire-deficient mice. Proceedings of the National Academy of Sciences, 104 (46) 18193-18198 doi:10.1073/pnas.0709160104

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Reference TypeJournal (article/letter/editorial)
TitleDanger-free autoimmune disease in Aire-deficient mice
JournalProceedings of the National Academy of Sciences
AuthorsGray, D. H. D.Author
Gavanescu, I.Author
Benoist, C.Author
Mathis, D.Author
Year2007 (November 13)Volume104
Issue46
PublisherProceedings of the National Academy of Sciences
DOIdoi:10.1073/pnas.0709160104Search in ResearchGate
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Mindat Ref. ID1744598Long-form Identifiermindat:1:5:1744598:6
GUID0
Full ReferenceGray, D. H. D., Gavanescu, I., Benoist, C., Mathis, D. (2007) Danger-free autoimmune disease in Aire-deficient mice. Proceedings of the National Academy of Sciences, 104 (46) 18193-18198 doi:10.1073/pnas.0709160104
Plain TextGray, D. H. D., Gavanescu, I., Benoist, C., Mathis, D. (2007) Danger-free autoimmune disease in Aire-deficient mice. Proceedings of the National Academy of Sciences, 104 (46) 18193-18198 doi:10.1073/pnas.0709160104
In(2007, November) Proceedings of the National Academy of Sciences Vol. 104 (46) Proceedings of the National Academy of Sciences

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Gavanescu, I., Benoist, C., Mathis, D. (2008) B cells are required for Aire-deficient mice to develop multi-organ autoinflammation: A therapeutic approach for APECED patients. Proceedings of the National Academy of Sciences, 105 (35) 13009-13014 doi:10.1073/pnas.0806874105
Gavanescu, I., Kessler, B., Ploegh, H., Benoist, C., Mathis, D. (2007) Loss of Aire-dependent thymic expression of a peripheral tissue antigen renders it a target of autoimmunity. Proceedings of the National Academy of Sciences, 104 (11) 4583-4587 doi:10.1073/pnas.0700259104
Feuerer, M., Jiang, W., Holler, P. D., Satpathy, A., Campbell, C., Bogue, M., Mathis, D., Benoist, C. (2007) Enhanced thymic selection of FoxP3+ regulatory T cells in the NOD mouse model of autoimmune diabetes. Proceedings of the National Academy of Sciences, 104 (46) 18181-18186 doi:10.1073/pnas.0708899104
Andre, I., Gonzalez, A., Wang, B., Katz, J., Benoist, C., Mathis, D. (1996) Checkpoints in the progression of autoimmune disease: lessons from diabetes models. Proceedings of the National Academy of Sciences, 93 (6) 2260-2263 doi:10.1073/pnas.93.6.2260
Guerau-de-Arellano, M., Mathis, D., Benoist, C. (2008) Transcriptional impact of Aire varies with cell type. Proceedings of the National Academy of Sciences, 105 (37) 14011-14016 doi:10.1073/pnas.0806616105
Flood, P. M., Benoist, C., Mathis, D., Murphy, D. B. (1986) Altered I-J phenotype in E alpha transgenic mice. Proceedings of the National Academy of Sciences, 83 (21) 8308-8312 doi:10.1073/pnas.83.21.8308
Gray, M., Miles, K., Salter, D., Gray, D., Savill, J. (2007) Apoptotic cells protect mice from autoimmune inflammation by the induction of regulatory B cells. Proceedings of the National Academy of Sciences, 104 (35) 14080-14085 doi:10.1073/pnas.0700326104
Venanzi, E. S., Melamed, R., Mathis, D., Benoist, C. (2008) The variable immunological self: Genetic variation and nongenetic noise in Aire-regulated transcription. Proceedings of the National Academy of Sciences, 105 (41) 15860-15865 doi:10.1073/pnas.0808070105
Tourne, S., Miyazaki, T., Wolf, P., Ploegh, H., Benoist, C., Mathis, D. (1997) Functionality of major histocompatibility complex class II molecules in mice doubly deficient for invariant chain and H-2M complexes. Proceedings of the National Academy of Sciences, 94 (17) 9255-9260 doi:10.1073/pnas.94.17.9255
Wang, B., Andre, I., Gonzalez, A., Katz, J. D., Aguet, M., Benoist, C., Mathis, D. (1997) Interferon-  impacts at multiple points during the progression of autoimmune diabetes. Proceedings of the National Academy of Sciences, 94 (25) 13844-13849 doi:10.1073/pnas.94.25.13844
Binstadt, B. A., Hebert, J. L., Ortiz-Lopez, A., Bronson, R., Benoist, C., Mathis, D. (2009) The same systemic autoimmune disease provokes arthritis and endocarditis via distinct mechanisms. Proceedings of the National Academy of Sciences, 106 (39) 16758-16763 doi:10.1073/pnas.0909132106
 
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