Carlson, G. C., Talbot, K., Halene, T. B., Gandal, M. J., Kazi, H. A., Schlosser, L., Phung, Q. H., Gur, R. E., Arnold, S. E., Siegel, S. J. (2011) Dysbindin-1 mutant mice implicate reduced fast-phasic inhibition as a final common disease mechanism in schizophrenia. Proceedings of the National Academy of Sciences, 108 (43) doi:10.1073/pnas.1109625108
Reference Type | Journal (article/letter/editorial) | ||
---|---|---|---|
Title | Dysbindin-1 mutant mice implicate reduced fast-phasic inhibition as a final common disease mechanism in schizophrenia | ||
Journal | Proceedings of the National Academy of Sciences | ||
Authors | Carlson, G. C. | Author | |
Talbot, K. | Author | ||
Halene, T. B. | Author | ||
Gandal, M. J. | Author | ||
Kazi, H. A. | Author | ||
Schlosser, L. | Author | ||
Phung, Q. H. | Author | ||
Gur, R. E. | Author | ||
Arnold, S. E. | Author | ||
Siegel, S. J. | Author | ||
Year | 2011 (October 25) | Volume | 108 |
Issue | 43 | ||
Publisher | Proceedings of the National Academy of Sciences | ||
DOI | doi:10.1073/pnas.1109625108Search in ResearchGate | ||
Generate Citation Formats | |||
Mindat Ref. ID | 1796295 | Long-form Identifier | mindat:1:5:1796295:1 |
GUID | 0 | ||
Full Reference | Carlson, G. C., Talbot, K., Halene, T. B., Gandal, M. J., Kazi, H. A., Schlosser, L., Phung, Q. H., Gur, R. E., Arnold, S. E., Siegel, S. J. (2011) Dysbindin-1 mutant mice implicate reduced fast-phasic inhibition as a final common disease mechanism in schizophrenia. Proceedings of the National Academy of Sciences, 108 (43) doi:10.1073/pnas.1109625108 | ||
Plain Text | Carlson, G. C., Talbot, K., Halene, T. B., Gandal, M. J., Kazi, H. A., Schlosser, L., Phung, Q. H., Gur, R. E., Arnold, S. E., Siegel, S. J. (2011) Dysbindin-1 mutant mice implicate reduced fast-phasic inhibition as a final common disease mechanism in schizophrenia. Proceedings of the National Academy of Sciences, 108 (43) doi:10.1073/pnas.1109625108 | ||
In | (2011, October) Proceedings of the National Academy of Sciences Vol. 108 (43) Proceedings of the National Academy of Sciences |
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