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Sojka, D. K., Fowell, D. J. (2011) Regulatory T cells inhibit acute IFN-  synthesis without blocking T-helper cell type 1 (Th1) differentiation via a compartmentalized requirement for IL-10. Proceedings of the National Academy of Sciences, 108 (45) 18336-18341 doi:10.1073/pnas.1110566108

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Reference TypeJournal (article/letter/editorial)
TitleRegulatory T cells inhibit acute IFN-  synthesis without blocking T-helper cell type 1 (Th1) differentiation via a compartmentalized requirement for IL-10
JournalProceedings of the National Academy of Sciences
AuthorsSojka, D. K.Author
Fowell, D. J.Author
Year2011 (November 8)Volume108
Issue45
PublisherProceedings of the National Academy of Sciences
DOIdoi:10.1073/pnas.1110566108Search in ResearchGate
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Mindat Ref. ID1796768Long-form Identifiermindat:1:5:1796768:2
GUID0
Full ReferenceSojka, D. K., Fowell, D. J. (2011) Regulatory T cells inhibit acute IFN-  synthesis without blocking T-helper cell type 1 (Th1) differentiation via a compartmentalized requirement for IL-10. Proceedings of the National Academy of Sciences, 108 (45) 18336-18341 doi:10.1073/pnas.1110566108
Plain TextSojka, D. K., Fowell, D. J. (2011) Regulatory T cells inhibit acute IFN-  synthesis without blocking T-helper cell type 1 (Th1) differentiation via a compartmentalized requirement for IL-10. Proceedings of the National Academy of Sciences, 108 (45) 18336-18341 doi:10.1073/pnas.1110566108
In(2011, November) Proceedings of the National Academy of Sciences Vol. 108 (45) Proceedings of the National Academy of Sciences

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Couzinet, A., Tamura, K., Chen, H.-m., Nishimura, K., Wang, Z., Morishita, Y., Takeda, K., Yagita, H., Yanai, H., Taniguchi, T., Tamura, T. (2008) A cell-type-specific requirement for IFN regulatory factor 5 (IRF5) in Fas-induced apoptosis. Proceedings of the National Academy of Sciences, 105 (7) 2556-2561 doi:10.1073/pnas.0712295105
Honda, K., Mizutani, T., Taniguchi, T. (2004) Negative regulation of IFN- /  signaling by IFN regulatory factor 2 for homeostatic development of dendritic cells. Proceedings of the National Academy of Sciences, 101 (8) 2416-2421 doi:10.1073/pnas.0307336101
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